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Mesenchymal neoplasm in breast

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Metrics details. Although adipocytes are the most abundant stromal cell component in breast cancer tissues, their interaction with breast cancer cells has been less investigated compared to cancer-associated fibroblasts or macrophages. Exosomes are a novel way of cell-cell communication and have been demonstrated to play an important role in various biological processes. However, to our knowledge, only a few studies have reported the effects of adipocyte exosomes on tumor development.
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Circulating breast tumor cells exhibit dynamic changes in epithelial and mesenchymal composition

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Mesenchymal tumors and tumor-like lesions of the breast: a contemporary approach review

A breast neoplasm is a growth in the breast that is abnormal in nature. Neoplasms are not necessarily malignant but usually diagnostic testing is needed to learn more about the type of growth and to develop an appropriate treatment plan. Patients may consult radiologists, oncologists, and other medical specialists in the process of diagnosing a neoplasm and deciding how they want to treat it, if treatment is required at all. Breast neoplasms can arise for a wide number of reasons.
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Malignant Mesenchymal Tumors

Mesenchyme is characterized morphologically by a prominent ground substance matrix containing a loose aggregate of reticular fibers and unspecialized mesenchymal stem cells. The mesenchyme originates from the mesoderm. This "soup" exists as a combination of the mesenchymal cells plus serous fluid plus the many different tissue proteins. Serous fluid is typically stocked with the many serous elements, such as sodium and chloride. The mesenchyme develops into the tissues of the lymphatic and circulatory systems, as well as the musculoskeletal system.
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Matrix metalloproteinases MMPs are essential for many physiological processes, but their inappropriate expression can facilitate the development and progression of tumors. Recognition of the relationship between matrix metalloproteinases and malignancy led to clinical trials of broad-spectrum MMP inhibitors as cancer therapeutics, but the results were disappointing. The failure of the clinical trials was due in large part to the propensity of the MMP inhibitors to inhibit essential physiological processes. Therapeutic strategies that target tumor-specific MMP -dependent effects may prove more promising. Previous studies and preliminary data show that exposure of mammary epithelial cells to selected matrix metalloproteinases causes cleavage of a cell surface molecule that stimulates cellular production of reactive oxygen species ROS.
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